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Anaphylaxis to 2 NSAIDs in a Patient Who Tolerated ASA.

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dc.contributor.authorBogas, G
dc.contributor.authorPerez-Sanchez, N
dc.contributor.authorAndreu, I
dc.contributor.authorDoña, I
dc.contributor.authorPerkins, J R
dc.contributor.authorBlanca, M
dc.contributor.authorCanto, G
dc.contributor.authorCornejo-Garcia, J A
dc.contributor.authorBlanca-Lopez, N
dc.contributor.funderEuropean Regional Development’s Funds (FEDER)
dc.contributor.funderCarlos III National Health Institute
dc.contributor.funderSpanish Ministry of Economy and Competitiveness
dc.contributor.funderRIRAAF
dc.contributor.funderAndalusian Health Service
dc.date.accessioned2023-01-25T08:34:49Z
dc.date.available2023-01-25T08:34:49Z
dc.date.issued2016
dc.description.abstractNonsteroidal anti-inflammatory drugs (NSAIDs) are the most frequent cause of drug hypersensitivity [1]. NSAID-induced reactions can be classified into 5 categories: (1) NSAID-exacerbated respiratory disease (NERD); (2) NSAID-exacerbated cutaneous disease (NECD); (3) NSAID-induced urticaria and/or angioedema (NIUA); (4) single NSAID-induced urticaria/angioedema/anaphylaxis (SNIUAA); and (5) single NSAID-induced delayed hypersensitivity reactions (SNIDR) [2]. Patients in the first 3 categories react to NSAIDs from various chemical groups in a nonallergic type of drug hypersensitivity known as cross-intolerance. The mechanism involved in these reactions is thought to be related to an imbalance in the arachidonic acid metabolic pathway leading to increased release of cysteinyl leukotrienes.
dc.description.sponsorshipJA Cornejo-García is funded by the Miguel Servet Program (Ref CP14/00034) and JR Perkins by the Sara Borrell Program (Ref CD14/00242) (Carlos III National Health Institute, Spanish Ministry of Economy and Competitiveness). This study was supported by grants from the Carlos III National Health Institute RD12/0013 (RIRAAF network), FIS (PI12/02247, PI13/02598, and PI15/00726), and the Andalusian Health Service (PI-0279-2012). This work was supported by grants from the European Regional Development’s Funds (FEDER).
dc.description.versionNo
dc.identifier.citationBogas G, Pérez-Sánchez N, Andreu I, Doña I, Perkins JR, Blanca M, et al. Anaphylaxis to 2 NSAIDs in a Patient Who Tolerated ASA. J Investig Allergol Clin Immunol. 2016;26(4):266-8
dc.identifier.doi10.18176/jiaci.0066
dc.identifier.issn1018-9068
dc.identifier.pmid27470646
dc.identifier.unpaywallURLhttp://www.jiaci.org/revistas/vol26issue4_7_2.pdf
dc.identifier.urihttp://hdl.handle.net/10668/10312
dc.issue.number4
dc.journal.titleJournal of investigational allergology & clinical immunology
dc.journal.titleabbreviationJ Investig Allergol Clin Immunol
dc.language.isoen
dc.organizationInstituto de Investigación Biomédica de Málaga-IBIMA
dc.organizationHospital Universitario Regional de Málaga
dc.page.number266-8
dc.provenanceRealizada la curación de contenido 10/03/2025
dc.publisherEsmon Publicidad
dc.pubmedtypeCase Reports
dc.pubmedtypeJournal Article
dc.pubmedtypeResearch Support, Non-U.S. Gov't
dc.relation.projectIDCP14/00034
dc.relation.projectIDCD14/00242
dc.relation.projectIDRD12/0013
dc.relation.projectIDPI12/02247
dc.relation.projectIDPI15/00726
dc.relation.projectIDPI-0279-2012
dc.relation.publisherversionhttp://www.jiaci.org/summary/vol26-issue4-num1383
dc.rights.accessRightsRestricted access
dc.subjectASA tolerance
dc.subjectAnaphylaxis
dc.subjectCross-intolerance
dc.subjectNSAID hypersensitivity
dc.subjectSelective reactions
dc.subjectHipersensibilidad a AINE
dc.subjectIntolerancia cruzada
dc.subjectReacciones selectivas
dc.subjectAnafilaxia
dc.subjectTolerancia a AAS
dc.subject.decsAntiinflamatorios no esteroideos
dc.subject.decsUrticaria
dc.subject.decsHipersensibilidad a las drogas
dc.subject.decsAngioedema
dc.subject.decsLeucotrienos
dc.subject.decsÁcido araquidónico
dc.subject.meshAnaphylaxis
dc.subject.meshAnti-Inflammatory Agents, Non-Steroidal
dc.subject.meshAspirin
dc.subject.meshHumans
dc.subject.meshMale
dc.subject.meshMiddle Aged
dc.titleAnaphylaxis to 2 NSAIDs in a Patient Who Tolerated ASA.
dc.typeresearch article
dc.type.hasVersionVoR
dc.volume.number26
dspace.entity.typePublication

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