%0 Journal Article
%A Valdivielso, P
%A Ramírez-Bollero, J
%A Pérez-López, C
%T Peripheral arterial disease, type 2 diabetes and postprandial lipidaemia: Is there a link?
%D 2014
%@ 1948-9358
%U http://hdl.handle.net/10668/1803
%X Peripheral arterial disease, manifested as intermittent claudication or critical ischaemia, or identified by an ankle/brachial index < 0.9, is present in at least one in every four patients with type 2 diabetes mellitus.Several reasons exist for peripheral arterial disease in diabetes. In addition to hyperglycaemia, smoking and hypertension, the dyslipidaemia that accompanies type2 diabetes and is characterised by increased triglyceride levels and reduced high-density lipoprotein cholesterol concentrations also seems to contribute to this association.Recent years have witnessed an increased interest in postprandial lipidaemia, as a result of various prospective studies showing that non-fasting triglyceridespredict the onset of arteriosclerotic cardiovascular disease better than fasting measurements do. Additionally, the use of certain specific postprandial particle markers, such as apolipoprotein B-48, makes it easier and more simple to approach the postprandial phenomenon. Despite this, only a few studies have evaluated the role ofpostprandial triglycerides in the development of peripheral arterial disease and type 2 diabetes. The purpose of this review is to examine the epidemiology and risk factors of peripheral arterial disease in type 2 diabetes, focusing on the role of postprandial triglycerides and particles.
%K Peripheral arterial disease
%K Type 2 diabetes
%K Postprandial lipidaemia
%K Apolipoprotein B-48
%K Anklebrachial index
%K Non-fasting triglycerides
%K Hiperlipidemias
%K Diabetes mellitus tipo 2
%K Enfermedad arterial periférica
%K HDL-Colesterol
%K LDL-Colesterol
%K Apolipoproteína B-48
%K Hemoglobina A Glucosilada
%K Triglicéridos
%K Índice tobillo braquial
%~