TY  - JOUR
AU  - Martinez-Losada, Carmen
AU  - Serrano-Lopez, Juana
AU  - Serrano-Lopez, Josefina
AU  - Noguera, Nelida I
AU  - Garza, Eduardo
AU  - Piredda, Liliana
AU  - Lavorgna, Serena
AU  - Consalvo, Maria Antonietta Irno
AU  - Ottone, Tiziana
AU  - Alfonso, Valentina
AU  - Peinado, Juan Ramon
AU  - Garcia-Ortiz, Maria Victoria
AU  - Morales-Ruiz, Teresa
AU  - Jerez, Andres
AU  - Hurtado, Ana Maria
AU  - Montesinos, Pau
AU  - Cervera, Jose
AU  - Such, Esperanza
AU  - Ibañez, Marian
AU  - Sempere, Amparo
AU  - Sanz, Miguel Angel
AU  - Lo-Coco, Francesco
AU  - Sanchez-Garcia, Joaquin
PY  - 2018
DO  - 10.3324/haematol.2018.188433
UR  - http://hdl.handle.net/10668/12316
T2  - Haematologica
AB  - Acute myeloid leukemia (AML) is a dynamic disease caused by accumulating, somatically acquired driver mutations generating branching competing clones.1 In favorable-risk AML, high resolution genomic profiling by single nucleotide polymorphism array...
LA  - en
PB  - Fondazione Ferrata Storti
KW  - Biomarkers, Tumor
KW  - Clonal Evolution
KW  - DNA Mutational Analysis
KW  - Genetic Association Studies
KW  - Genome-Wide Association Study
KW  - Humans
KW  - Leukemia, Myeloid, Acute
KW  - Mutation
KW  - Nuclear Proteins
KW  - Nucleophosmin
KW  - Phenotype
KW  - Prognosis
KW  - Recurrence
TI  - Clonal genetic evolution at relapse of favorable-risk acute myeloid leukemia with NPM1 mutation is associated with phenotypic changes and worse outcomes.
TY  - research article
VL  - 103
ER  -