TY  - JOUR
AU  - Rivera, Patricia
AU  - Silva-Peña, Daniel
AU  - Blanco, Eduardo
AU  - Vargas, Antonio
AU  - Arrabal, Sergio
AU  - Serrano, Antonia
AU  - Pavón, Francisco Javier
AU  - Bindila, Laura
AU  - Lutz, Beat
AU  - Rodríguez de Fonseca, Fernando
AU  - Suárez, Juan
PY  - 2018
DO  - 10.1016/j.neuropharm.2018.11.037
UR  - http://hdl.handle.net/10668/13259
T2  - Neuropharmacology
AB  - Previous findings demonstrate a homeostatic role for oleoylethanolamide (OEA) signaling in the ethanol-related neuroinflammation and behavior. However, extensive research is still required in order to unveil the effects of OEA on a number of...
LA  - en
KW  - Alcohol
KW  - Locomotion
KW  - Microglia
KW  - Neurogenesis
KW  - PPARα
KW  - Striatum
KW  - Alanine Transaminase
KW  - Alcohol Drinking
KW  - Amidohydrolases
KW  - Animals
KW  - Apoptosis
KW  - Arachidonic Acids
KW  - Aspartate Aminotransferases
KW  - Calcium-Binding Proteins
KW  - Caspase 3
KW  - Cell Proliferation
KW  - Cell Survival
KW  - Endocannabinoids
KW  - Ethanol
KW  - Ethanolamines
KW  - Glial Fibrillary Acidic Protein
KW  - Hepatobiliary Elimination
KW  - Locomotion
KW  - Male
KW  - Microfilament Proteins
KW  - Microglia
KW  - Neostriatum
KW  - Neurons
KW  - Oleic Acids
KW  - PPAR alpha
KW  - Phospholipase D
KW  - Polyunsaturated Alkamides
KW  - Proto-Oncogene Proteins c-fos
KW  - Rats
KW  - Rats, Wistar
KW  - Signal Transduction
KW  - gamma-Glutamyltransferase
TI  - Oleoylethanolamide restores alcohol-induced inhibition of neuronal proliferation and microglial activity in striatum.
TY  - research article
VL  - 146
ER  -