TY  - JOUR
AU  - Aragones, Gemma
AU  - Dasuri, Kalavathi
AU  - Olukorede, Opeoluwa
AU  - Francisco, Sarah G
AU  - Renneburg, Carol
AU  - Kumsta, Caroline
AU  - Hansen, Malene
AU  - Kageyama, Shun
AU  - Komatsu, Masaaki
AU  - Rowan, Sheldon
AU  - Volkin, Jonathan
AU  - Workman, Michael
AU  - Yang, Wenxin
AU  - Daza, Paula
AU  - Ruano, Diego
AU  - Dominguez-Martin, Helena
AU  - Rodriguez-Navarro, Jose Antonio
AU  - Du, Xue-Liang
AU  - Brownlee, Michael A
AU  - Bejarano, Eloy
AU  - Taylor, Allen
PY  - 2020
DO  - 10.1111/acel.13257
UR  - http://hdl.handle.net/10668/16541
T2  - Aging cell
AB  - Diabetes and metabolic syndrome are associated with the typical American high glycemia diet and result in accumulation of high levels of advanced glycation end products (AGEs), particularly upon aging. AGEs form when sugars or their metabolites react...
LA  - en
PB  - Wiley-Blackwell Publishing Ltd.
KW  - aging
KW  - autophagy
KW  - glycative stress
KW  - p62
KW  - proteotoxicity
KW  - Animals
KW  - Autophagy
KW  - Cell Line
KW  - Cell Survival
KW  - Epithelial Cells
KW  - Glycation End Products, Advanced
KW  - Humans
KW  - Kidney
KW  - Lens, Crystalline
KW  - Lysosomes
KW  - Mice
KW  - Mice, Inbred C57BL
KW  - Mice, Knockout
KW  - RNA-Binding Proteins
KW  - Rats
TI  - Autophagic receptor p62 protects against glycation-derived toxicity and enhances viability.
TY  - research article
VL  - 19
ER  -