TY  - JOUR
AU  - Parras, Alberto
AU  - Anta, Hector
AU  - Santos-Galindo, Maria
AU  - Swarup, Vivek
AU  - Elorza, Ainara
AU  - Nieto-Gonzalez, Jose L
AU  - Pico, Sara
AU  - Hernandez, Ivo H
AU  - Diaz-Hernandez, Juan I
AU  - Belloc, Eulalia
AU  - Rodolosse, Annie
AU  - Parikshak, Neelroop N
AU  - Peñagarikano, Olga
AU  - Fernandez-Chacon, Rafael
AU  - Irimia, Manuel
AU  - Navarro, Pilar
AU  - Geschwind, Daniel H
AU  - Mendez, Raul
AU  - Lucas, Jose J
PY  - 2018
DO  - 10.1038/s41586-018-0423-5
UR  - http://hdl.handle.net/10668/12840
T2  - Nature
AB  - Common genetic contributions to autism spectrum disorder (ASD) reside in risk gene variants that individually have minimal effect sizes. As environmental factors that perturb neurodevelopment also underlie idiopathic ASD, it is crucial to identify...
LA  - en
PB  - Nature Publishing Group
KW  - Autism spectrum disorders
KW  - Translation
KW  - Animals
KW  - Autism Spectrum Disorder
KW  - Brain
KW  - Exons
KW  - Female
KW  - Genetic Predisposition to Disease
KW  - Humans
KW  - Male
KW  - Mice
KW  - Mice, Transgenic
KW  - Neurons
KW  - Phenotype
KW  - Polyadenylation
KW  - Protein Binding
KW  - RNA Splicing
KW  - RNA, Messenger
KW  - RNA-Binding Proteins
KW  - Transcriptome
TI  - Autism-like phenotype and risk gene mRNA deadenylation by CPEB4 mis-splicing.
TY  - research article
VL  - 560
ER  -