TY  - JOUR
AU  - Pintado-Berninches, Laura
AU  - Fernandez-Varas, Beatriz
AU  - Benitez-Buelga, Carlos
AU  - Manguan-Garcia, Cristina
AU  - Serrano-Benitez, Almudena
AU  - Iarriccio, Laura
AU  - Carrillo, Jaime
AU  - Guenechea, Guillermo
AU  - Egusquiaguirre, Susana P
AU  - Pedraz, Jose-Luis
AU  - Hernández, Rosa M
AU  - Igartua, Manoli
AU  - Arias-Salgado, Elena G
AU  - Cortés-Ledesma, Felipe
AU  - Sastre, Leandro
AU  - Perona, Rosario
PY  - 2019
DO  - 10.1038/s41418-018-0272-7
UR  - http://hdl.handle.net/10668/13445
T2  - Cell death and differentiation
AB  - Ataxia telangiectasia (AT) is a genetic disease caused by mutations in the ATM gene but the mechanisms underlying AT are not completely understood. Key functions of the ATM protein are to sense and regulate cellular redox status and to transduce DNA...
LA  - en
KW  - Ataxia Telangiectasia
KW  - Ataxia Telangiectasia Mutated Proteins
KW  - Cell Cycle Proteins
KW  - Cell Line
KW  - DNA Breaks, Double-Stranded
KW  - DNA Damage
KW  - Fibroblasts
KW  - Humans
KW  - Nanoparticles
KW  - Nuclear Proteins
KW  - Oxidative Stress
KW  - Peptide Fragments
KW  - Phosphorylation
KW  - Reactive Oxygen Species
KW  - Telomerase
KW  - Telomere
TI  - GSE4 peptide suppresses oxidative and telomere deficiencies in ataxia telangiectasia patient cells.
TY  - research article
VL  - 26
ER  -