TY  - JOUR
AU  - Colagrossi, Luna
AU  - Hermans, Lucas E
AU  - Salpini, Romina
AU  - Di Carlo, Domenico
AU  - Pas, Suzan D
AU  - Alvarez, Marta
AU  - Ben-Ari, Ziv
AU  - Boland, Greet
AU  - Bruzzone, Bianca
AU  - Coppola, Nicola
AU  - Seguin-Devaux, Carole
AU  - Dyda, Tomasz
AU  - Garcia, Federico
AU  - Kaiser, Rolf
AU  - Köse, Sukran
AU  - Krarup, Henrik
AU  - Lazarevic, Ivana
AU  - Lunar, Maja M
AU  - Maylin, Sarah
AU  - Micheli, Valeria
AU  - Mor, Orna
AU  - Paraschiv, Simona
AU  - Paraskevis, Dimitros
AU  - Poljak, Mario
AU  - Puchhammer-Stöckl, Elisabeth
AU  - Simon, François
AU  - Stanojevic, Maja
AU  - Stene-Johansen, Kathrine
AU  - Tihic, Nijaz
AU  - Trimoulet, Pascale
AU  - Verheyen, Jens
AU  - Vince, Adriana
AU  - Lepej, Snjezana Zidovec
AU  - Weis, Nina
AU  - Yalcinkaya, Tülay
AU  - Boucher, Charles A B
AU  - Wensing, Annemarie M J
AU  - Perno, Carlo F
AU  - Svicher, Valentina
AU  - HEPVIR working group of the European Society for translational antiviral research (ESAR)
PY  - 2018
DO  - 10.1186/s12879-018-3161-2
UR  - http://hdl.handle.net/10668/12535
T2  - BMC infectious diseases
AB  - HBsAg immune-escape mutations can favor HBV-transmission also in vaccinated individuals, promote immunosuppression-driven HBV-reactivation, and increase fitness of drug-resistant strains. Stop-codons can enhance HBV oncogenic-properties. Furthermore,...
LA  - en
KW  - Drug-resistance
KW  - HBV
KW  - HBsAg
KW  - Immune-escape
KW  - Stop-codons
KW  - Adult
KW  - Amino Acid Substitution
KW  - Antiviral Agents
KW  - Codon, Terminator
KW  - Europe
KW  - Female
KW  - Genotype
KW  - Hepatitis B Surface Antigens
KW  - Hepatitis B virus
KW  - Hepatitis B, Chronic
KW  - Humans
KW  - Male
KW  - Middle Aged
KW  - Mutation
TI  - Immune-escape mutations and stop-codons in HBsAg develop in a large proportion of patients with chronic HBV infection exposed to anti-HBV drugs in Europe.
TY  - research article
VL  - 18
ER  -