TY  - JOUR
AU  - Caballano-Infantes, Estefanía
AU  - Garcia-Garcia, Alberto
AU  - Lopez-Gomez, Carlos
AU  - Cueto, Alejandro
AU  - Robles-Diaz, Mercedes
AU  - Ortega-Alonso, Aida
AU  - Martin-Reyes, Flores
AU  - Alvarez-Alvarez, Ismael
AU  - Arranz-Salas, Isabel
AU  - Ruiz-Cabello, Francisco
AU  - Lucena, Isabel M
AU  - Garcia-Fuentes, Eduardo
AU  - Andrade, Raul J
AU  - Garcia-Cortes, Miren
PY  - 2021
DO  - 10.3390/biomedicines10010055
SN  - 2227-9059
UR  - http://hdl.handle.net/10668/20811
T2  - Biomedicines
AB  - Non-alcoholic fatty liver disease (NAFLD) and idiosyncratic drug-induced liver injury (DILI) could share molecular mechanisms involving the immune system. We aimed to identify activation immunological biomarkers in invariant natural killer T (iNKT)...
LA  - en
PB  - MDPI AG
KW  - drug-induced liver injury (DILI)
KW  - immune response
KW  - immunophenotype
KW  - liver fibrosis
KW  - non-alcoholic fatty liver disease (NAFLD)
KW  - Humans
KW  - Non-alcoholic Fatty Liver Disease
KW  - NK Cell Lectin-Like Receptor Subfamily K
KW  - Natural Killer T-Cells
KW  - Insulin Resistance
KW  - HLA-DR Antigens
KW  - Liver Cirrhosis
KW  - Biomarkers
KW  - CD8-Positive T-Lymphocytes
KW  - Chemical and Drug Induced Liver Injury
KW  - Aspartate Aminotransferases
KW  - Killer Cells, Natural
TI  - Differential iNKT and T Cells Activation in Non-Alcoholic Fatty Liver Disease and Drug-Induced Liver Injury.
TY  - research article
VL  - 10
ER  -