TY  - JOUR
AU  - Cobos, Enrique J
AU  - Nickerson, Chelsea A
AU  - Gao, Fuying
AU  - Chandran, Vijayendran
AU  - Bravo-Caparros, Inmaculada
AU  - Gonzalez-Cano, Rafael
AU  - Riva, Priscilla
AU  - Andrews, Nick A
AU  - Latremoliere, Alban
AU  - Seehus, Corey R
AU  - Perazzoli, Gloria
AU  - Nieto, Francisco R
AU  - Joller, Nicole
AU  - Painter, Michio W
AU  - Ma, Chi Him Eddie
AU  - Omura, Takao
AU  - Chesler, Elissa J
AU  - Geschwind, Daniel H
AU  - Coppola, Giovanni
AU  - Rangachari, Manu
AU  - Woolf, Clifford J
AU  - Costigan, Michael
PY  - 2018
DO  - 10.1016/j.celrep.2018.01.006
UR  - http://hdl.handle.net/10668/12070
T2  - Cell reports
AB  - Chronic neuropathic pain is a major morbidity of neural injury, yet its mechanisms are incompletely understood. Hypersensitivity to previously non-noxious stimuli (allodynia) is a common symptom. Here, we demonstrate that the onset of cold...
LA  - en
PB  - Cell Press
KW  - T cells
KW  - TrpV1
KW  - WCGNA
KW  - cold allodynia
KW  - gene expression
KW  - immune system
KW  - macrophages
KW  - neuropathic pain
KW  - tactile allodynia
KW  - transcript profiling
KW  - Animals
KW  - Behavior, Animal
KW  - Cold Temperature
KW  - Hyperalgesia
KW  - Macrophages
KW  - Male
KW  - Mice
KW  - Mice, Inbred C57BL
KW  - Mice, Transgenic
KW  - Neuralgia
KW  - Sensory Receptor Cells
KW  - T-Lymphocytes
KW  - TRPV Cation Channels
KW  - Touch
KW  - Transcriptome
TI  - Mechanistic Differences in Neuropathic Pain Modalities Revealed by Correlating Behavior with Global Expression Profiling.
TY  - research article
VL  - 22
ER  -